By A. Claudio Cuello
The necessity for potent remedy to regard Alzheimer’s ailment is bigger than ever, yet there's nonetheless no drug treatment which could cease or opposite the development of the disorder. there's, even though, loads of anticipation over the upcoming improvement of powerful remedies as a result identity of promising pursuits for drug improvement. This ebook investigates those ambitions and examines ongoing thoughts to increase powerful cures for this devastating neurodegenerative situation.
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Extra info for Pharmacological Mechanisms in Alzheimer's Therapeutics
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C. Cuello & B. ), Pharmacological sciences: perspectives for research and therapy in the late 1990s (pp. 241–254). Basel: Birkhauser. , et al. (1994). Apolipoprotein E-4 gene dose in clinically diagnosed Alzheimer’s disease: Prevalence, plasma cholesterol levels and cerebrovascular change. European Archives of Psychiatry and Clinical Neuroscience, 243, 291–292. , & Maloney, A. J. (1976). Selective loss of central cholinergic neurons in Alzheimer’s disease. Lancet, 2, 1403. De, R. , Garcia, A. , et al.
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Prada, C. , et al. (1996). Increased amyloid-beta42(43) in brains of mice expressing mutant presenilin 1. Nature, 383, 710–713. , Grant, S. , et al. (2004). Altered mitogen-activated protein kinase signaling, tau hyperphosphorylation and mild spatial learning dysfunction in transgenic rats expressing the betaamyloid peptide intracellularly in hippocampal and cortical neurons. Neuroscience, 129, 583–592. Eckman, E. , & Eckman, C. B. (2005). Abeta-degrading enzymes: Modulators of Alzheimer’s disease pathogenesis and targets for therapeutic intervention.
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