By Philip M. Parker
In March 2001, the nationwide Institutes of well-being issued the subsequent caution: ''The variety of sites supplying health-related assets grows each day. Many websites offer useful details, whereas others could have details that's unreliable or misleading.'' additionally, due to the speedy elevate in Internet-based info, many hours should be wasted looking out, opting for, and printing. when you consider that merely the smallest fraction of knowledge facing hemochromatosis is listed in se's, akin to www.google.com or others, a non-systematic method of net learn will be not just time eating, but additionally incomplete. This publication used to be created for doctors, scholars, and individuals of most of the people who are looking to behavior scientific study utilizing the main complex instruments on hand and spending the smallest amount of time doing so.
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Extra resources for Hemochromatosis - A Bibliography and Dictionary for Physicians, Patients, and Genome Researchers
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The ferroportin1 gene is defective in the zebrafish mutant weissherbst. Weissherbst embryos die from severe iron deficiency anemia, resulting from defective iron transport between the yolk sac and the developing embryo. The mammalian ortholog of ferroportin1 is expressed in the basolateral membrane of intestinal enterocytes, and at other sites requiring active iron expert. We hypothesize that ferroportin1 is the basolateral iron transporter in enterocytes and an iron exporter in other cell types In this grant we propose to study the conservation of vertebrate ferroportin1 function and the role of ferroportin1 in vivo.
We believe our preliminary data have answered the first two questions in the affirmative. This proposal focuses on the third question and has two specific aims: 1) to devise methods to increase iron accumulation in mouse epidermis a) genetically by creating a transgenic mouse that overexpresses the transferrin receptor in epidermis b) pharmacologically by topical application of nitrosopine, a derivative of nifedipine. 2) to test whether these methods of increasing iron in epidermis are able to reduce the iron burden in a mouse model of iron overload a) by breeding the transgenic, transferrin receptor overexpressor mouse to Hfe null mice; b) by topical application of nitrosopine, a derivative of nifedipine, to Hfe null mice.
Professor of Medicine; Pediatrics; Columbia University Health Sciences Columbia University Medical Center New York, Ny 100323702 Timing: Fiscal Year 2005; Project Start 30-SEP-2001; Project End 30-APR-2007 Summary: (provided by applicant): The proposed Bioengineering Research Partnership will integrate bioengineering, basic science and clinical efforts in the design, development and clinical validation of a high-transition-temperature (high Tc; operating at 77oK) superconducting susceptometer for the direct, non-invasive measurement of hepatic iron stores in patients with iron overload from hereditary hemochromatosis, thalassemia major, sickle cell disease and other disorders.