By David H. Spodick
Offers up to date insurance of the pericardium in well-being and clinical and surgical illness, integrating contemporary advances in knowing general and irregular pericardial body structure and immunology, acute and protracted pericarditis, effusion and tamponade, and imaging equipment for effusion and constriction. accommodates scientific, hemodynamic, investigative, and experimental paintings performed via best interdisciplinary researchers--including never-before-published reports--on the traditional and diseased pericardium.
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Additional info for The Pericardium: A Comprehensive Textbook (Fundamental and Clinical Cardiology)
Example text
Nephrology provided frequent acute uremic and infectious pericarditis in all their clinical and hemodynamic phases and later the new, recalcitrant "dialysis pericarditis," while improved survival now permitted the emergence of occasional uremic constriction. Urgent pericardiocentesis was required when new patients' first dialyses rapidly decreased blood volume, precipitating previously compensated or occult cardiac tamponade. This pericardial pandemic inspired my first book, Acute Pericarditis, followed by Chronic and Constrictive Pericarditis.
Independent, detailed exposition is also give to electrocardiography (ECG) (Chapter 5) because it is a test that is widely familiar and often at least basically understood among physicians of all kinds. Furthermore, on a practical level, certain major as well as finer points of ECG that are important in pericardial diseases tend to get massed by most current computer programs. Because thrombolysis has provoked hemopericardium in patients misdiagnosed as having acute infarction, there is heavy emphasis on "diagnostic" EGCs as well as typical and atypical variants with many illustrations.
Limitation of hypertrophy associated with chronic exerciseMembranous functions I. Reduction of external friction due to heart movements. A. Production of pericardial fluid. B. Generation of phospholipid surfactants. 1 II. Buttressing of thinner portions of the myocardium (reciprocal variations in parietal pericardial thickness). A. Atria. B. Right ventricle. III. Defensive immunologic constituents in pericardial fluid. IV. Fibrinolytic activity in mesothelial lining. V. Prostacyclin (PGE2, PGI2 and eicosanoids) released into pericardial sac in response to stretch, hypoxia and increased myocardial loading/work.