By Sunil Nadar, Gregory Lip
The administration of high blood pressure varies significantly from clinician to clinician. This pocketbook is a concise and evidence-based precis of present knowing and perform, together with the main updated tips from nationwide our bodies and their ideas for high blood pressure care.
An crucial software for daily use in dealing with high blood pressure, this booklet covers the various levels of research and definitive administration, and aids scientific decision-making via collating all of the suitable details and suggestions in a single simply obtainable position.
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Example text
Safian RD and Textor SC (200). Renal-artery stenosis. New England Journal of Medicine; Feb; 344(6): 43–42. , Lerman L. (20). State of the art: Renovascular hypertension and ischaemic nephropathy. American Journal of hypertension; 23: 59. Working Group on Renovascular Hypertension. (987). Detection, evaluation, and treatment of renovascular hypertension. Final report. Working Group on Renovascular Hypertension. Archives of Internal Medicine; May; 47(5): 820–9. 19 Chapter 4 Primary hyperaldosteronism Sunil Nadar Key points • Primary hyperaldosteronism is an important treatable cause of hypertension.
Working Group on Renovascular Hypertension. Archives of Internal Medicine; May; 47(5): 820–9. 19 Chapter 4 Primary hyperaldosteronism Sunil Nadar Key points • Primary hyperaldosteronism is an important treatable cause of hypertension. • It is caused by aldosterone-producing tumours. • Screening would be beneficial in patients with resistant hypertension and young hypertensive patients. • Serum potassium alone as a screening test for primary hyperaldosteronism would miss nearly two-thirds of patients.
Of patients with hypertension have biochemical results that indicate the need for investigations for PHA. Bendroflumethiazide and related thiazide diuretics in the low doses used nowadays usually do not cause hypokalaemia. Thus, a significant fall in potassium with thiazides may reflect underlying aldosterone excess. Although spontaneous hypokalaemia in a patient with hypertension is a strong indicator that hyperaldosteronism is present, at least 20% of hypertensive patients have a low normal serum potassium level and this therefore is not useful as a diagnostic tool.