By Jr. Cooper
Leslie T. Cooper, Jr., MD, and a panel of prime medical and clinical specialists comprehensively evaluate either advances in easy clinical figuring out and present medical wisdom of inflammatory middle illness. at the scientific facet they talk about the analysis, analysis, and remedy of nonspecific and particular myocarditis in separate chapters dedicated to such significant idiopathic scientific entities as cardiac sarcoidosis, mammoth phone myocarditis, eosinophilic myocarditis, Chagas ailment, rheumatic fever, and human immunodeficiency virus-related cardiomyopathy. study articles whole the image with discussions of the position of apoptosis in viral myocarditis, rat and mouse types of autoimmune great telephone and lymphocytic myocarditis, and the most recent on hand info on enteroviral proteases and cardiomyopathy.
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169. 170. 171. 172. 173. feron-γ protects mice from lethal coxsackievirus B3 infection and subsequent myocarditis. Nat Med 2000;6:693-697. Tu Z, Chapman NM, Hufnagel G, Tracy S, Romero JR, Barry WH, Zhao L, Currey K, Shapiro B. The cardiovirulent phenotype of coxsackievirus B3 is determined at a single site in the genomic 5′ nontranslated region. J Virol 1995;69:4607-4618. Dunn JJ, Chapman NM, Tracy S, Romero JR. Genomic determinants of cardiovirulence in coxsackievirus B3 clinical isolates: localization to the 5′ nontranslated region.
Current data, however, do indicate that CAR is both necessary and sufficient for CVB infection of cells on which it is expressed, either naturally or after transfection. 63,66 Consequently, we speculate that DAF and CAR may be closely associated on the cell surface, that DAF may serve to provide a cell-associated reservoir of virus thereby increasing access to CAR, and that the optimally functional CVB receptor may be a complex of proteins in which CAR is the principal and key component. Resolution of this issue awaits detailed structural analysis of CVB bound to CAR, to DAF, and to the putative DAFCAR complex and molecular description of the participation of these proteins in virus binding and cell entry.
112. Traystman MD, Beisel KW. Genetic control of coxsackievirus B3-induced heart-specific autoantibodies associated with chronic myocarditis. Clin Exp Immunol 1991;86:291-298. 113. Huber SA, Lodge PA. Coxsackievirus B-3 myocarditis. Identification of different pathogenic mechanisms in DBA/2 and Balb/c mice. Am J Pathol 1986;122:284-291. 114. Huber SA, Weller A, Herzum M, Lodge PA, Estrin M, Simpson K, Guthrie M. Immunopathogenic mechanisms in experimental picornavirus-induced autoimmunity. Pathol Immunopathol Res 1988;7:279-291.