By Christopher P. Cannon
During this moment version of his hugely acclaimed publication, Christopher P. Cannon, MD, and a workforce of preeminent medical investigators have completely revised and multiplied each bankruptcy to mirror the hot explosion of medical trials at the administration of acute coronary syndromes (ACS). This comprises: the more moderen exams, comparable to C reactive protein; a multimarker method of prognosis and probability stratification; new antiplatelet brokers; and mixtures of thrombolytic-antithrombotic regimens. additionally mentioned are the newest advancements in interventional cardiology, using cost-effectiveness in scientific trials to decide on acceptable cures, and the employment of severe pathways research to enhance compliance with evidence-based medication and instruction innovations.
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Additional info for Management of Acute Coronary Syndromes (Contemporary Cardiology)
Sample text
Effects of platelet glycoprotein IIb/IIIa receptor blockade by a chimeric monoclonal antibody (abciximab) on acute and six-month outcomes after percutaneous transluminal coronary agioplasty for acute myocardial infarction. Am J Cardiol 1996;77:1045–1051. 123. Montalescot G, Barragan P, Wittenberg O, et al. Platelet glycoprotein IIb/IIIa inhibition with coronary stenting for acute myocardial infarction. N Engl J Med 2001;344:1895–1903. 124. Stone GW, Grines CL, Cox DA, et al. Comparison of angioplasty with stenting, with or without abciximab, in acute myocardial infarction.
2. The modulation of coagulation is a vital component of natural vascular thromboresistance. Protein C (PC) binds to surface thrombomodulin (TM) and, in the presence of protein S (PS), forms C APC, which then neutralizes two coagulation proteases—factor V and factor VIII. TFPI, antithrombin (AT), heparin cofactor II (HCII), and annexin V are also important components of thromboresistance. It has become apparent that hypercholesterolemia adversely effects endothelial cell function (even before the development of atherosclerosis), and although morphologically intact, the vascular endothelium in regions of atherosclerosis fails to release nitric oxide (13).
Time to reperfusion: the critical modulator in thrombolysis and primary angioplasty. J Thromb Thrombolysis 1996;3:117–125. 101. Garcia-Dorado D, Theroux P, Tornos P, et al. Previous aspirin use may attenuate the severity of the manifestation of acute ischemic syndromes. Circulation 1995;92:1743–1748. 102. Borzak S, Cannon CP, Kraft PL, et al. Effects of prior aspirin and anti-ischemic therapy on outcome of patients with unstable angina. Am J Cardiol 1998;81:678–681. 103. Willard JE, Lange RA, Hillis LD.