By Jong S. Kim, Louis R. Caplan, K. S. Lawrence Wong

Intracranial atherosclerosis is the dominant explanation for stroke in over 70% of the world’s inhabitants. Globalization is resulting in an more and more heterogeneous society far and wide. Advances in imaging techinology permit this formerly inaccessible pathology to be clinically studied.Edited by way of the world over well known clinicians, Intracranial Atherosclerosis is the 1st booklet to envision intracranial reasons of stroke. scientific perform is allied with simple technology to lead all people with an curiosity in stroke at the prognosis and administration of intracranial atherosclerosis.

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In addition, there are other methods to produce cerebral atherosclerosis in animals. 44 successfully developed fat deposition in the posterior communicating arteries in normotensive rats, which were fed high-fat cholesterol (HFC) for 10 weeks after bilateral or unilateral carotid artery ligation or basilar artery ligation. These findings suggest that not only high blood pressure but also hemodynamic derangements are important factors for the development of fat deposition in cerebral arteries. 45 Intimal injury was produced by a silicone rubber cylinder with nylon thread that was injected from the bifurcation of the right external and internal carotid arteries to embolize either to the intracranial internal carotid or middle cerebral artery.

The PCA most often then extends posteriorly to spread over the ipsilateral cortex, whereas the proximal connection with the PCoA regresses. In this common scenario, the primary arterial supply shifts to a source from the terminal basilar artery. 9 Digital subtraction angiography illustrating posterior cerebral artery anastomosis with posterior communicating artery, thalamoperforators, and distal cortical branches. from what has been termed a fetal PCoA. 59 The PCA extends from the terminal portion of the basilar artery in the interpeduncular cistern, passing above the oculomotor (III) nerve to circle the midbrain above the tentorium (Fig.

Acta Anat (Basle) 1982; 113: 108–116. Brandt T, von Kummer R, Muller-Kuppers M, Hacke W. Thrombolytic therapy of acute basilar artery occlusion. Variables affecting recanalization and outcome. Stroke 1996; 27: 875–881. Zeal AA. and Rhoton AL, Jr. Microsurgical anatomy of the posterior cerebral artery. J Neurosurg 1978; 48: 534– 559. Bisaria KK. Anomalies of the posterior communicating artery and their potential clinical significance. J Neurosurg 1984; 60: 572–576. Milisavljevic MM, Marinkovic SV, Gibo H, Puskas LF.

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