By Robert J Amato

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7, (May 2010), pp. 277-285. ; Bratslavsky, G. ; Pinto, P. ; Schmidt, L. ; Neckers, L. ; Bottaro, D. & Srinivasan, R. (2010b). Molecular Diagnosis and Therapy of Kidney Cancer. 61, pp. 329-343. ; Montironi, R. ; Egevad, L. ; Caballero-Vargas, M. ; Scarpelli, M. ; Kirkali, Z. & Cheng, L. (2010). Genetic Profiles in Renal Tumors. 17, pp. 6-19. ; Harries, R. (1990). Clinical Features and Natural History of von HippelLindau Disease. 77, pp. 1151-1163. ; Chang, G. (1999). The Tumour Suppressor Protein VHL Targets Hypoxia-inducible Factors for Oxygen-dependent Proteolysis.

30-34. ; Epstein, J. & Sesterhenn, I. (2004). Pathology and Genetics. Tumors of the Urinary System and Male Genital Organs. IARC Press, Lyon, France. ; Delgado, M. & Roach, E. (1998). Renal Lesion Growth in Children with Tuberous Sclerosis Complex. 1, pp. 141–145. ; Linehan, W. & Merino, M. (1998). Collecting Duct Carcinomas of the Kidney: a Comparative Loss of Heterozygosity Study with Clear Cell Renal Cell Carcinoma. Oncol. 5, pp. 923–926. ; Cober, M. & Mittermayer, C. (1992). Collecting Duct Carcinoma: Cytogenetic Characterization.

4991–5004. , Lechpammer, M. & Kaelin, W. G. Jr. (2002). Inhibition of HIF is Necessary for Tumor Suppression by the von Hippel–Lindau Protein. 1, pp. 237–246. ; Beckwith, B. (1997). The Heidelberg Classification of Renal Cell Tumours. 131-133. ; Schnell, P. (2003). Von Hippel-Lindau Protein Binds Hyperphosphorylated Large Subunit of RNA Polymerase II Through a Proline Hydroxylation Motif and Targets it for Ubiquitination. 100, pp. 2706-2711. ; Lee, E. (2004). A Distinct Expression Pattern and Point Mutation of c-kit in Papillary Renal Cell Carcinomas.

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