By June Halper, Nancy J. Holland
Textual content on offering nursing care to people with MS, for nursing practitioners and scholars. Addresses the actual, emotional, social, and environmental affects of the disorder. This variation comprises chapters at the nursing implications, complimentary treatments, and sufferer and kinfolk schooling. earlier version: c1997. Softcover. DNLM: a number of Sclerosis--nursing.
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Additional resources for Comprehensive Nursing Care in Multiple Sclerosis 2nd Edition
In the absence of evidence for a specific viral trigger for MS, increased attention has been paid to the possibility that many viruses and bacteria— and perhaps other pathogens as well—could serve as triggers for the disease process. If so, it may well be the manner in which a genetically susceptible immune system reacts to such infectious challenges that determines if MS will occur. This possibility decreases the likelihood that a vaccine against a specific triggering agent could be found to prevent disease.
There is evidence from population and family studies that HLA genes in people with MS differ from healthy individuals. This is not as straighforward as it seems: HLA genetic makeup can differ between MS populations, independent of health status, and thus there is no clear-cut “MS HLA gene profile” that defines the entire MS population. A second important gene type hypothesized to be involved in immune function in MS is the gene responsible for the structure and function of receptors on T-cells of the immune system.
DEMYELINATION: THE CAUSE OF SYMPTOMS The damage done by an immune reaction against myelin and nerve fibers in brain and spinal cord might truly be considered the “cause” of MS since it is this process that results in symptoms. Immune system T-cells normally in the blood stream become activated against a myelin antigen and cross the blood–brain barrier, which separates the blood stream from the CNS and normally keeps such immune system cells out of the nervous system. Local inflammation in scattered places—most often around blood vessels and ventricles of the brain—results in damage to the myelin insulation around nerve fibers.
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