By William Montagna, Richard L. Dobson
Advances in Biology of dermis, quantity VII: Carcinogenesis covers court cases of the fifteenth Symposium at the Biology of epidermis, held on the Oregon local Primate examine middle on April 9/11, 1965, lower than the auspices of the college of Oregon clinical institution.
This publication consists of 19 chapters, and starts off with the mechanism of tissue homeostasis in grownup mammals and the kinetics of epidermal response to carcinogenic brokers and different epidermis irritants. The succeeding chapters take care of the expansion selling results of tumors on tissues, the response trend differences among general and neoplastic epithelium, and a few organic implications of chemical carcinogenesis. enormous chapters are dedicated to quite a few cancer causing agents, together with hydrocarbons, viruses, androgens, and estrogens. different chapters contemplate the physicochemical mechanisms of acceleration of epidermis carcinogenesis and experimental observations of environmental carcinogenesis. The mechanisms of pores and skin melanoma induction as a result of ultraviolet radiation, in addition to arsenic caused tumors are tested. The concluding chapters describe a few varieties of dermis tumors, corresponding to adnexal tumors and basal mobile epithelioma.
This e-book will turn out necessary to oncologists and researchers within the box of carcinogenesis.
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Additional resources for Carcinogenesis. Proceedings of a Symposium on the Biology of Skin Held at the University of Oregon Medical School, 1965
Path. 4, 245-265. BRENT, T. , BUTLER, J. A. V. and CRATHORN, A. R. (1965). Variations in phosphokinase activities during the cell cycle in synchronous populations of HeLa cells. Nature 207, 176-177. BÛCHER, N . L. R. (1963). Regeneration of mammalian liver. Int. Rev. Cytol. 15, 245-300. BÛCHER, N . L. , SCOTT, J. F . and A U B , J. C. (1951). Regeneration of the liver in parabiotic rats. Cancer Res. 11, 457-465. BÛCHER, N . L. R. and SWAFFIELD, M. N . (1964). The rate of incorporation of labelled thymidine into the deoxyribonucleic acid of regenerating rat liver in relation to the amount of liver excised.
A combination of these factors can also occur. From our observations we think that the dynamics of the hyperplasia after a single application of carcinogens and non-carcinogenic hyperplasia-producing skin irritants are more or less the same. But this similarity does not apply to the continuous application of carcinogens. If the alteration of the cell population kinetics after methylcholanthrene is compared with that provoked by cantharidin, we see that cantharidin can produce a pronounced hyperplasia characterized by a high rate of cell proliferation, a high mitotic count, and a low mitotic duration.
J. VAN and EKEL, T. M. (1963). Kinetics of hyperplasia in psoriasis. Arch. Derm. 88, 373-381. SCOTT, R. B. and BELL, E. (1964). Protein synthesis during development: control through messenger RNA. Science 145, 711-713. SEKERIS, C. E. and LANG, N . (1964). Stimulation of messenger R N A synthesis in rat liver by Cortisol. Life Sei. 3, 169-174. SETÄLÄ, K. (1965). Differences in pharmacodynamic response to colchicine between benign and malignant epidermal hyperplasias. , suppl. 237,1-89. SHERMAN, F .
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