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This can be a 3-in-1 reference publication. It supplies an entire clinical dictionary masking hundreds of thousands of phrases and expressions when it comes to atherosclerosis. It additionally offers broad lists of bibliographic citations. ultimately, it presents info to clients on easy methods to replace their wisdom utilizing a variety of web assets. The ebook is designed for physicians, clinical scholars getting ready for Board examinations, scientific researchers, and sufferers who are looking to get to grips with learn devoted to atherosclerosis. in the event that your time is efficacious, this booklet is for you. First, you won't waste time looking out the net whereas lacking loads of correct info. moment, the ebook additionally saves you time indexing and defining entries. ultimately, you won't waste time and cash printing hundreds of thousands of web content.
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Extra resources for Atherosclerosis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
Example text
We now propose to better define the in utero programming associated with maternal hypercholesterolemia, to identify genes influencing post-natal susceptibility to atherosclerosis, and to investigate whether interventions during pregnancy also decrease the susceptibility in offspring of "normocholesterolemic" mothers. Different levels of maternal hypercholesterolemia and antioxidant protection will be achieved by transferring embryos of LDL receptor deficient (LDLR-/-) mice into treated or untreated C57BL/6, LDLR -/- or apoE -/- mice.
For twelve cases, the angiographic and IVUS records will be used to describe the 3-D lumen and wall of a portion of the vessel in diastole. The flow fieled and wall stresses in this region will be simulated numerically, using the Doppler and pressure data as input; the first flow calculation will be validated experimentally. By comparing the computed distribution of stresses acting on and in the vessel segment against that of intima/media thickness, inferences will be drawn regarding the role of mechanical forces in relation to human atherosclerosis.
Etiology of Atherosclerosis: Lipoproteins and other Risk Factors (hypertension, diabetes, homocystinemia); 2. Lesion Initiation; 3. Inflammation; 4. Foam Cell Formation and Regression; 5. Complexity of Lesions; 6. Plaque Rupture and Thrombosis; 7. Genomic Approaches; 8. Intervention and Diagnosis; 9. The Genome Project and Vascular Disease. The conference will be a major vehicle for the integration of new knowledge in the field of atherosclerosis research. In order to stimulate an interdisciplinary dialogue, speakers will be invited who represent distinct but overlapping areas of research such as hypertension, genomics, lipoproteins and vascular biology.